Why is Singapore's haze back in 2026 and what do PM2.5 readings mean for health?
Haze in Southeast Asia is a recurring seasonal problem linked to asthma, heart, mental, nerve and eye harm, but the sources do not confirm the cause, timing or severity of the 2026 Singapore episode.
Covers: This page explains the likely causes of the 2026 haze episode in Singapore, including regional land and peat fires and weather patterns, and translates PM2.5 air quality readings into practical health guidance. It does not provide real-time forecasts or personal medical advice.
Also answers: Why is there haze in Singapore again in 2026? · What does PM2.5 mean for my health in Singapore? · Singapore haze 2026 causes and PM2.5 health effects · Is the haze back in Singapore in 2026?
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The short answer
Interpretation AI-prepared starting mapThe sources here establish that transboundary haze in Southeast Asia is a recurring seasonal problem with measurable acute health effects, but none of them confirms the specific cause, timing or severity of the 2026 haze episode in Singapore. What the evidence does show: asthma emergency department visits in Singapore rise with the Pollutant Standards Index (same-day correlation r=0.142, 95% CI 0.107–0.178), and mean PSI is higher during haze (82.67 vs 51.46). In southern Thailand, PM2.5 above 50 µg/m³ was linked to asthma exacerbations with odds ratios of 1.41–1.64 versus the lowest exposure group. In Singapore, every 10 µg/m³ rise in PM2.5 was associated with a 3.8% cumulative increase in conjunctivitis risk, with a disproportionate burden during haze episodes. Regional reviews link haze to respiratory, cardiovascular, psychological and neurological harm, and estimate up to 296,000 annual deaths from wildfire smoke during El Niño events.12345
- Evidence 16
- Interpretation 4
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Be the first to voteIn brief
In Singapore, asthma emergency department visits rise with PSI on the same day of exposure, and mean PSI is markedly higher during haze (82.67 vs 51.46).1
Evidence-backedPM2.5 above 50 µg/m³ was associated with 1.41–1.64 times the odds of asthma exacerbation compared with the lowest exposure group, and children aged 6–17 were more susceptible even at 15–25 µg/m³.2
Evidence-backedEvery 10 µg/m³ increase in PM2.5 was associated with a 3.8% cumulative rise in conjunctivitis risk in Singapore, with a disproportionate burden during haze episodes.3
Evidence-backedNo source in this set confirms the specific cause, timing or measured air quality of the 2026 episode; that remains unconfirmed.
Interpretation
At a glance
The picture in numbers
Live · updated just now
- During haze82.67 PSI
- Outside haze51.46 PSI
3.8%
4 in every 100
296,000 deaths
1.41–1.64
times the odds
The high estimate is 1.2 times the low one.
The evidence behind it
5 sources- Reviews of many studies2
- Other studies and data3
When it was published
Newest from 2026
| Source | Kind | Year |
|---|---|---|
| Association between weather, air quality and asthma-related emergency department visits: a retrospective time-series study in Singapore. | Other studies and data | 2025 |
| Attributing climate-related health burdens in Southeast Asia: a scoping review. | Reviews of many studies | 2026 |
| Short-term exposure to fine particulate matter and asthma exacerbation: a large population-based case-crossover study in Southern Thailand. | Other studies and data | 2025 |
| Acute Health Impacts of the Southeast Asian Transboundary Haze Problem—A Review | Reviews of many studies | 2019 |
| The burden of acute conjunctivitis attributable to ambient particulate matter pollution in Singapore and its exacerbation during South-East Asian haze episodes | Other studies and data | 2020 |
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What it means for you
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Pick the situation closest to yours. Each answer says what it rests on.
If you have asthma or another respiratory condition
the evidence links higher PSI and PM2.5 to more emergency department visits and exacerbations, with the strongest association on the same day of exposure, so same-day awareness of readings is the relevant signal.12
Evidence-backedIf you are caring for a child aged 6 to 17
this age group showed greater susceptibility to asthma exacerbations, including at PM2.5 levels of 15–25 µg/m³, which is below the level that raised risk in the wider population.2
Evidence-backedIf you are prone to eye irritation or conjunctivitis
each 10 µg/m³ rise in PM2.5 was associated with a 3.8% cumulative increase in conjunctivitis risk over the present and following week, and that burden was disproportionately higher during haze episodes.3
Evidence-backedIf you want to know whether a given day's air is likely to affect you
the Singapore asthma data showed the association peaked on the same day of exposure, so current-day readings are more informative than readings from several days earlier.1
Evidence-backedThe full story · 4 chapters
01
What is known about the 2026 episode
AI summary:No source here confirms the cause, timing or measured air quality of the 2026 Singapore episode; only the general seasonal haze mechanism is established.
Interpretation: None of the sources provided describes the 2026 haze episode in Singapore directly. There is no confirmed report here of the fire locations, the weather pattern involved, or the measured air quality during the episode. Readers should treat any specific causal claim about 2026 as unconfirmed until a source addresses it.
Evidence-backed: What the sources do establish is the general mechanism: transboundary haze in Southeast Asia is a seasonal problem, and the 2019 review describes it as recurrent but intermittent exposure to high levels of seasonal haze. The 2026 scoping review links wildfire smoke during El Niño events to an estimated 296,000 annual deaths across the region, indicating that El Niño conditions are a recognised driver of severe haze years.54
How concerned are you about the health effects of haze on you or your family?
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02
What PM2.5 and PSI readings mean
AI summary:PM2.5 is fine inhalable particulate matter, PSI is Singapore's composite index, and higher readings track higher conjunctivitis and asthma risk.
Evidence-backed: PM2.5 refers to fine particulate matter small enough to be inhaled deep into the lungs. The Singapore conjunctivitis study quantified the risk gradient: every 10 µg/m³ increase in PM2.5 was associated with a 3.8% cumulative increase in conjunctivitis risk over the present and subsequent week (IRR 1.038, 95% CI 1.029–1.046). Every 10 µg/m³ increase in the coarser PM10 was associated with a 2.9% increase (IRR 1.029, 95% CI 1.022–1.036). About 3% of all acute conjunctivitis reports were attributable to PM2.5, and that attributable burden was disproportionately higher during transboundary haze episodes.3
Evidence-backed: The Pollutant Standards Index (PSI) is the composite index used in Singapore. In the Singapore asthma study, mean PSI was 82.67 during haze versus 51.46 outside it. The southern Thailand study used a different metric — raw PM2.5 concentration — and found that concentrations above 50 µg/m³ were significantly associated with asthma exacerbations at multiple time lags (lag0, lag2, and lag01 to lag03), with odds ratios of 1.41 to 1.64 compared with the lowest group (0–15 µg/m³). That study also found children aged 6–11 and 12–17 were more susceptible, with elevated risk even at the lower band of 15–25 µg/m³.12
03
Health effects linked to haze exposure
AI summary:Haze exposure is linked to asthma visits, eye irritation, and respiratory, cardiovascular, psychological and neurological harm.
Evidence-backed: Respiratory effects are the best documented. In Singapore, asthma emergency department visits were positively correlated with PSI at lag 0 (r=0.142, 95% CI 0.107–0.178) and inversely correlated with rainfall at lag 3 (r=-0.062), wet-bulb temperature at lag 1 (r=-0.067), wind speed at lag 2 (r=-0.049) and ambient temperature at lag 6 (r=-0.045). The association peaked on the same day of exposure but was no longer significant after adjusting for demographic and clinical factors. The authors also associated pollution-linked festivals, transboundary haze and the Northeast monsoon with increased asthma ED visits.1
Evidence-backed: Beyond asthma, the 2019 review reports consistent links between haze exposure and acute psychological, respiratory, cardiovascular and neurological morbidity and mortality across countries affected by transboundary haze. The 2026 scoping review adds that air pollution research is linked to up to 31% of all-cause mortality in the studies it examined, and that heat — the second most studied exposure — was associated with up to 8.61% of mortality in Bangkok.54
Evidence-backed: Eye irritation is a less commonly discussed but quantified effect: the Singapore conjunctivitis study found particulate matter exposure increased the risk of conjunctival disease, with a disproportionately higher disease burden during Southeast Asian transboundary haze episodes.3
04
Who appears most susceptible
AI summary:Children aged 6 to 17 appear more susceptible to asthma exacerbations, even at lower PM2.5 levels.
Evidence-backed: Children stand out in the evidence. The southern Thailand case-crossover study found children aged 6–11 and 12–17 exhibited greater susceptibility to asthma exacerbations, particularly at PM2.5 concentrations of 15–25 µg/m³ — a band well below the >50 µg/m³ threshold that raised risk in the overall population. Associations at early lags were seen for both males and females.2
Evidence-backed: The Singapore asthma study adjusted for demographic, clinical and socioeconomic factors, implying these modify the pollution–asthma relationship, though the source does not report which subgroups were most affected. The 2019 review notes that the mechanism by which these particles cause disease and mortality still needs further investigation.15
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What to remember
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PM2.5 above µg/m³ was associated with 1.41–1.64 times the odds of asthma exacerbation compared with the lowest exposure group, and children aged 6–17 were more susceptible even at 15–25 µg/m³.
Every µg/m³ increase in PM2.5 was associated with a 3.8% cumulative rise in conjunctivitis risk in Singapore, with a disproportionate burden during haze episodes.
Transboundary haze is a recurring seasonal problem in Southeast Asia with consistent links to acute respiratory, cardiovascular, psychological, neurological and eye-related harm.
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- 1Association between weather, air quality and asthma-related emergency department visits: a retrospective time-series study in Singapore.BMJ open (Toh et al.)Published Dec 18, 2025Checked Oct 12, 2026
“Associations between ED visits and monsoons, transboundary haze and cultural festivals were evaluated using one-way analysis of variance. Weekly seasonal ARIMA models with exogenous regressors were fitted, incorporating PSI as a covariate and adjusting for demographic, clinical and socioeconomic factors.ResultsAsthma ED visits were positively correlated with PSI (lag 0: r=0.142; 95% CI 0.107 to 0.178) and inversely correlated with rainfall (lag 3: r=-0.062; 95% CI -0.099 to -0.026) and WBT (lag 1: r=-0.067; 95% CI -0.104 to -0.031). Wind speed (lag 2: r=-0.049; 95% CI -0.086 to -0.013) and ambient temperature (lag 6: r=-0.045; 95% CI -0.081 to -0.008) showed weaker inverse associations. Mean PSI was higher during haze (82.67 vs 51.46, pConclusionsPSI-ED association peaked on the same day of exposure but was no longer significant after adjusting for demographic and clinical factors. Pollution-linked festivals, transboundary haze and the Northeast monsoon were associated with increased asthma ED visits.”
- 2Short-term exposure to fine particulate matter and asthma exacerbation: a large population-based case-crossover study in Southern Thailand.Environmental health : a global access science source (Varopichetsan et al.)Published May 7, 2025Checked Oct 12, 2026
“PM2.5 concentrations > 50 µg/m³ were significantly associated with asthma exacerbations at multiple time lags (lag0, lag2, and lag01 to lag03), with odds ratios ranging from 1.41 to 1.64, compared to the lowest concentration group (PM2.5 0-15 µg/m³). Temperature showed no significant effect, while relative humidity was positively associated with asthma exacerbations at lag3, lag06, and lag07. Subgroup analyses revealed associations between PM2.5 exposure and asthma exacerbations at early lags for both males and females. Additionally, children aged 6-11 years and 12-17 years exhibited greater susceptibility to asthma exacerbations, particularly at PM2.5 concentrations of 15-25 µg/m³.ConclusionThis study underscores the short-term effects of PM2.5 on asthma exacerbations, particularly during high-pollution episodes of transboundary haze in regions that generally experience low levels of air pollution. These findings emphasize the importance of achieving the WHO air quality targets to mitigate the health impacts from PM2.5.”
- 3The burden of acute conjunctivitis attributable to ambient particulate matter pollution in Singapore and its exacerbation during South-East Asian haze episodesThe Science of The Total Environment (Aik et al.)Published Jun 11, 2020Checked Oct 12, 2026
“Every 10 μg/m3 increase in PM2.5 was associated with a 3.8% (Incidence Rate Ratio (IRR): 1.038, 95% Confidence Interval (CI): 1.029–1.046, p < 0.001) cumulative increase in risk of conjunctivitis over the present and subsequent week. Every 10 μg/m3 increase in PM10 was associated with a 2.9% (Incidence Rate Ratio (IRR): 1.029, 95% Confidence Interval (CI): 1.022–1.036, p < 0.001) cumulative increase in risk of conjunctivitis over the present and subsequent week. Acute conjunctivitis reports exhibited an inverse dependence on ambient air temperature and relative humidity variability. Approximately 3% of all acute conjunctivitis reports were attributable to PM2.5. Particulate matter attributed acute conjunctivitis was disproportionately higher during transboundary haze episodes. Our study strengthens the evidence linking particulate matter exposure to an increased risk of conjunctival disease, with a disproportionately higher disease burden during South-East Asia transboundary haze episodes. Our findings underscore the importance of reducing the health impact of indigenous and transboundary sources of ambient particulate matter pollution.”
- 4Attributing climate-related health burdens in Southeast Asia: a scoping review.The Lancet regional health. Western Pacific (Simion et al.)Published Aug 21, 2026Checked Oct 12, 2026
“Although growing evidence demonstrates substantial health impacts from air pollution, heat, and extreme weather, the regional research landscape remains critically imbalanced, marked by neglect of other major hazards-including floods, droughts, extreme rainfall, storms and sea-level rise-and substantial methodological fragmentation. From 5294 records screened, 77 studies met our inclusion criteria. The evidence is dominated by air pollution research, the focus of 70% of studies and linked to up to 31% of all-cause mortality and an estimated 296,000 annual deaths from wildfire smoke during El Niño events. Heat, the second most studied exposure, was associated with up to 8.61% of mortality in Bangkok. In contrast, extreme events such as heatwaves, floods and storms were least frequently investigated. These findings reveal a critical misalignment between regional climate risks and current research focus. Standardized attribution frameworks are urgently needed to support integrated climate-health modeling and guide equitable, evidence-based adaptation across Southeast Asia.”
- 5Acute Health Impacts of the Southeast Asian Transboundary Haze Problem—A ReviewInternational Journal of Environmental Research and Public Health (Cheong et al.)Published Sep 6, 2019Checked Oct 12, 2026
“In light of growing public health concerns, recent studies have begun to examine the health effects of this seasonal haze problem in Southeast Asia. This review paper aims to synthesize current research efforts on the impact of the Southeast Asian transboundary haze on acute aspects of public health. Existing studies conducted in countries affected by transboundary haze indicate consistent links between haze exposure and acute psychological, respiratory, cardiovascular, and neurological morbidity and mortality. Future prospective and longitudinal studies are warranted to quantify the long-term health effects of recurrent, but intermittent, exposure to high levels of seasonal haze. The mechanism, toxicology and pathophysiology by which these toxic particles contribute to disease and mortality should be further investigated. Epidemiological studies on the disease burden and socioeconomic cost of haze exposure would also be useful to guide policy-making and international strategy in minimizing the impact of seasonal haze in Southeast Asia.”
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Open questions
What specifically caused the 2026 haze episode — which land or peat fires, in which locations, and under what weather pattern? No source here addresses this.
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What were the actual PM2.5 and PSI readings recorded in Singapore during the 2026 episode, and how do they compare with the exposure levels studied in the research cited here?
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What are the long-term health effects of recurrent but intermittent exposure to high seasonal haze, given that the 2019 review found these remain unquantified?
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Which specific protective actions (masks, air purifiers, staying indoors, timing of outdoor activity) have been shown to reduce the health effects quantified in these studies?
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