Why is Singapore's haze back and what do PM2.5 readings mean for health?
Haze in Singapore comes back each dry season from land-clearing fires, and studies link it to more asthma visits and child pneumonia.
Covers: The causes of recurring transboundary haze episodes in Singapore, how to read PSI and PM2.5 figures, and what research says about health effects of fine particulate exposure. Does not give personal medical advice or real-time air quality forecasts.
Also answers: Why is the haze back in Singapore? · What does PM2.5 mean for my health? · Singapore haze PSI PM2.5 health effects · Is the haze in Singapore dangerous?
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The short answer
Interpretation AI-prepared starting mapSingapore's haze is a recurring transboundary air pollution problem caused mainly by slash-and-burn land clearing for agriculture (notably palm oil and pulpwood), which flares up each dry season and is generally worst between July and October and during El Niño events; severe episodes occurred in 1997 and 2015. On health, Singapore data show asthma emergency department visits rise with the Pollutant Standards Index (PSI), peaking on the same day of exposure, though the association weakened after adjusting for demographic and clinical factors. Regional studies link fine particles (PM2.5) to asthma exacerbations and childhood pneumonia, with the strongest risks during high-pollution episodes.1234
- Evidence 18
- Interpretation 2
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Be the first to voteIn brief
Singapore's haze is a recurring transboundary problem driven mainly by slash-and-burn land clearing, worst between July and October and during El Niño years.1
Evidence-backedIn Singapore, asthma emergency department visits rise with PSI, peaking on the same day, though the link weakened after adjusting for demographic and clinical factors.2
Evidence-backedPM2.5 above 50 µg/m³ was linked to asthma exacerbations with odds ratios of 1.41–1.64, and children were more susceptible even at 15–25 µg/m³.3
Evidence-backedFine particles (PM2.5) are strongly linked to childhood pneumonia, while PM10 is more closely tied to acute respiratory infection.4
Evidence-backedRegional mortality estimates for smoke haze vary enormously, from about 1,064 to 260,000 deaths depending on assumptions.5
Evidence-backed
At a glance
The picture in numbers
Live · updated just now
- During haze periods82.67 PSI
- Outside haze periods51.46 PSI
- Low estimate1,064 deaths
- All-cause, all ages100,000 deaths
- High estimate260,000 deaths
0–15 µg/m³
lowest exposure group
15–25 µg/m³
children more susceptible
above 50 µg/m³
significantly linked to exacerbations
The evidence behind it
5 sources- Reviews of many studies1
- Other studies and data3
- Background1
When it was published
Newest from 2026
| Source | Kind | Year |
|---|---|---|
| Association between weather, air quality and asthma-related emergency department visits: a retrospective time-series study in Singapore. | Other studies and data | 2025 |
| Climate Change Adaptation to Smoke Haze for Improved Child Health in Southeast Asia: Analysis Situation in Palembang, Indonesia. | Other studies and data | 2026 |
| Short-term exposure to fine particulate matter and asthma exacerbation: a large population-based case-crossover study in Southern Thailand. | Other studies and data | 2025 |
| A scoping review on the health effects of smoke haze from vegetation and peatland fires in Southeast Asia: Issues with study approaches and interpretation. | Reviews of many studies | 2022 |
| Southeast Asian haze (Wikipedia) | Background | Unknown |
The community around it
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What it means for you
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If you have asthma or another respiratory condition
note that Singapore data show asthma emergency department visits rise with PSI on the same day, and that PM2.5 above 50 µg/m³ was linked to asthma exacerbations with odds ratios of 1.41–1.64 in a regional study.23
Evidence-backedIf you are caring for children
be aware that children aged 6–17 showed greater susceptibility to asthma exacerbations at PM2.5 of 15–25 µg/m³, and that childhood pneumonia has been linked to PM2.5 rises during haze.34
Evidence-backedIf you are planning around the haze season
expect episodes to be worst between July and October and during El Niño events, based on the historical pattern of transboundary haze since 1972.1
Evidence-backedIf you want to interpret a PSI reading
remember that the Singapore asthma study used PSI as its exposure measure and found mean PSI of 82.67 during haze versus 51.46 outside it, so PSI is the metric with local health-association data.2
Evidence-backedIf you are looking for a single number for haze deaths in the region
treat any figure cautiously, as estimates range from about 1,064 to 260,000 depending on cause, age group, study area and period.5
Evidence-backedThe full story · 4 chapters
01
Why the haze keeps coming back
AI summary:Haze is recurring transboundary pollution from slash-and-burn land clearing, worst July to October and in El Niño years, with 1997 and 2015 severe.
Evidence-backed: The Southeast Asian haze is a fire-related, recurrent transboundary air pollution issue. It is caused primarily by slash-and-burn land clearing, and the problem flares up every dry season to varying degrees, generally worst between July and October and during El Niño events. Transboundary haze in the region has been recorded since 1972, with the 1997 and 2015 events being particularly severe.1
Evidence-backed: Industrial-scale slash-and-burn to clear land for agriculture is a major cause, particularly for palm oil and pulpwood production. Burning is used because it is cheaper and faster than cutting and clearing with machinery. Fires started for this purpose sometimes spread and become forest fires, worsening the problem, and the high concentration of peat in the soil contributes to the haze's density and high sulphur content.1
02
Reading PSI and PM2.5
AI summary:PSI summarises Singapore air quality, while PM2.5 is the fine fraction most linked to respiratory harm, especially pneumonia in children.
Evidence-backed: Singapore's air quality is commonly summarised by the Pollutant Standards Index (PSI). In a Singapore time-series study of asthma emergency department visits, mean PSI was higher during haze periods (82.67) than outside them (51.46). That study used PSI as its exposure covariate, so it speaks to PSI specifically rather than to PM2.5 concentrations in isolation.2
Evidence-backed: PM2.5 — fine particulate matter — is the fraction most often linked to respiratory harm because fine particles penetrate deeper into the respiratory tract. In a study of childhood respiratory illness in Palembang, Indonesia, PM2.5 showed strong associations with pneumonia, while the coarser PM10 was more consistently associated with acute respiratory infection (ARI). Sulphur dioxide associations were less consistent, though it may act synergistically with particulate matter.4
03
What the research says about health effects
AI summary:Asthma visits rose with same-day PSI but the link weakened after adjustment; PM2.5 above 50 µg/m³ was tied to asthma flare-ups, and children were more vulnerable.
Evidence-backed: In Singapore, asthma emergency department visits were positively correlated with PSI at lag 0 (same-day exposure), with r=0.142 (95% CI 0.107 to 0.178). Visits were inversely correlated with rainfall (lag 3: r=-0.062), wet-bulb temperature (lag 1: r=-0.067), wind speed (lag 2: r=-0.049) and ambient temperature (lag 6: r=-0.045). However, the PSI–ED association was no longer significant after adjusting for demographic and clinical factors. Pollution-linked festivals, transboundary haze and the Northeast monsoon were associated with increased asthma ED visits.2
Evidence-backed: In Southern Thailand, a large population-based case-crossover study found that PM2.5 concentrations above 50 µg/m³ were significantly associated with asthma exacerbations at multiple time lags (lag0, lag2, and lag01 to lag03), with odds ratios of 1.41 to 1.64 compared with the lowest concentration group (0–15 µg/m³). Children aged 6–11 and 12–17 were more susceptible, particularly at PM2.5 of 15–25 µg/m³. Temperature showed no significant effect, while relative humidity was positively associated with exacerbations at lag3, lag06 and lag07.3
Evidence-backed: A scoping review of smoke haze from vegetation and peatland fires in Southeast Asia found that health burden estimation studies put deaths attributable to smoke haze across the whole region at approximately 100,000 when considering all-cause mortality and all age groups, with estimates ranging from 1,064 to 260,000 depending on the specified mortality cause, age group, study area and study period. Health risk assessment studies quantified potential lifetime cancer and non-cancer risks from smoke-related chemicals. All approaches revealed potential health risks, but the review notes a lack of interconnection between them.5
Evidence-backed: Children appear especially vulnerable. In Palembang, Indonesia, haze exposure significantly elevated the risk of respiratory illness in children, with regression models indicating that rises in PM2.5 predicted higher pneumonia incidence. The review authors argue that the persistent nature of haze and children's heightened vulnerability mean child-centred risk mitigation should be integrated into national air quality management and disaster preparedness.4
04
What to watch as episodes develop
AI summary:Haze timing is broadly predictable, and risks may appear even at moderate PM2.5 levels in low-pollution places like Singapore.
Interpretation: Because haze is seasonal and tied to dry-season burning and El Niño, the timing of episodes is broadly predictable even if individual days are not. The Southern Thailand study's finding that risks are elevated even at moderate PM2.5 levels (15–25 µg/m³) in regions that generally experience low air pollution suggests that places like Singapore may see health effects at concentrations below the most severe thresholds.31
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What to remember
Try to recall each hidden figure before you reveal it. Remembering, not rereading, is what makes it stick.
PM2.5 above µg/m³ was linked to asthma exacerbations with odds ratios of 1.41–1.64, and children were more susceptible even at 15–25 µg/m³.
Regional mortality estimates for smoke haze vary enormously, from about deaths depending on assumptions.
Singapore's haze is a recurring transboundary problem driven mainly by slash-and-burn land clearing, worst between July and October and during El Niño years.
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- 1Southeast Asian haze (Wikipedia)WikipediaPublished Oct 10, 2026Checked Oct 10, 2026
“The Southeast Asian haze is a fire-related recurrent transboundary air pollution issue. Haze events, where air quality reaches hazardous levels due to high concentrations of airborne particulate matter from burning biomass, have caused adverse health, environmental and economic impacts in several countries in Southeast Asia. Caused primarily by slash-and-burn land clearing, the problem flares up every dry season to varying degrees and generally is worst between July and October and during El Niño events. Transboundary haze in Southeast Asia has been recorded since 1972 with the 1997 and 2015 events being particularly severe. Industrial-scale slash-and-burn practices to clear land for agricultural purposes are a major cause of the haze, particularly for palm oil and pulpwood production in the region. Burning land occurs as it is cheaper and faster compared to cutting and clearing using excavators or other machinery. Fires started for this purpose sometimes spread and create forest fires, worsening the problem. The high concentration of peat in soil contributes to the haze's density and high sulphur content.”
- 2Association between weather, air quality and asthma-related emergency department visits: a retrospective time-series study in Singapore.BMJ open (Toh et al.)Published Dec 18, 2025Checked Oct 10, 2026
“Associations between ED visits and monsoons, transboundary haze and cultural festivals were evaluated using one-way analysis of variance. Weekly seasonal ARIMA models with exogenous regressors were fitted, incorporating PSI as a covariate and adjusting for demographic, clinical and socioeconomic factors.ResultsAsthma ED visits were positively correlated with PSI (lag 0: r=0.142; 95% CI 0.107 to 0.178) and inversely correlated with rainfall (lag 3: r=-0.062; 95% CI -0.099 to -0.026) and WBT (lag 1: r=-0.067; 95% CI -0.104 to -0.031). Wind speed (lag 2: r=-0.049; 95% CI -0.086 to -0.013) and ambient temperature (lag 6: r=-0.045; 95% CI -0.081 to -0.008) showed weaker inverse associations. Mean PSI was higher during haze (82.67 vs 51.46, pConclusionsPSI-ED association peaked on the same day of exposure but was no longer significant after adjusting for demographic and clinical factors. Pollution-linked festivals, transboundary haze and the Northeast monsoon were associated with increased asthma ED visits.”
- 3Short-term exposure to fine particulate matter and asthma exacerbation: a large population-based case-crossover study in Southern Thailand.Environmental health : a global access science source (Varopichetsan et al.)Published May 7, 2025Checked Oct 10, 2026
“PM2.5 concentrations > 50 µg/m³ were significantly associated with asthma exacerbations at multiple time lags (lag0, lag2, and lag01 to lag03), with odds ratios ranging from 1.41 to 1.64, compared to the lowest concentration group (PM2.5 0-15 µg/m³). Temperature showed no significant effect, while relative humidity was positively associated with asthma exacerbations at lag3, lag06, and lag07. Subgroup analyses revealed associations between PM2.5 exposure and asthma exacerbations at early lags for both males and females. Additionally, children aged 6-11 years and 12-17 years exhibited greater susceptibility to asthma exacerbations, particularly at PM2.5 concentrations of 15-25 µg/m³.ConclusionThis study underscores the short-term effects of PM2.5 on asthma exacerbations, particularly during high-pollution episodes of transboundary haze in regions that generally experience low levels of air pollution. These findings emphasize the importance of achieving the WHO air quality targets to mitigate the health impacts from PM2.5.”
- 4Climate Change Adaptation to Smoke Haze for Improved Child Health in Southeast Asia: Analysis Situation in Palembang, Indonesia.Annals of global health (Haryanto et al.)Published Jun 8, 2026Checked Oct 10, 2026
“Particulate matter showed strong associations with increased childhood respiratory morbidity. Pneumonia demonstrated more consistent relationships with PM2.5, reflecting the deeper penetration of fine particles into the respiratory tract. Regression models indicated that rises in PM2.5 predicted higher pneumonia incidence, while PM10 was more consistently associated with ARI. Associations involving SO2 were less consistent, though it may act synergistically with particulate matter to worsen respiratory outcomes. Haze exposure significantly elevates the risk of respiratory illness in children. PM2.5 is strongly linked to pneumonia, whereas PM10 is more closely related to ARI. The persistent nature of haze and children's heightened vulnerability highlight the need to integrate child-centered risk mitigation into national air quality management and disaster preparedness strategies.”
- 5A scoping review on the health effects of smoke haze from vegetation and peatland fires in Southeast Asia: Issues with study approaches and interpretation.PloS one (Phung et al.)Published Sep 15, 2022Checked Oct 10, 2026
“mpared to non-smoke haze periods; HBE studies estimated approximately 100,000 deaths attributable to smoke haze in the entire Southeast Asia considering all-cause mortality and all age groups, which ranged from 1,064-260,000 for specified mortality cause, age group, study area, and study period; HRA studies quantified potential lifetime cancer and non-cancer risks due to exposure to smoke-related chemicals. Currently, there is a lack of interconnection between these three approaches. The EPI approach requires extensive effort to investigate lifetime health effects, whereas the HRA approach needs to clarify the assumptions in exposure assessments to estimate lifetime health risks. The HBE approach allows the presentation of health impact in different scenarios, however, the risk functions used are derived from EPI studies from other regions. Two recent studies applied a combination of the EPI and HBE approaches to address uncertainty issues due to the selection of risk functions. In conclusion, all approaches revealed potential health risks due to smoke haze. Nonetheless, future studies should consider comparable exposure assessments to allow the integration of the three approaches.”
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Published 1 time since Oct 10, 2026.
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“Why the haze keeps coming back” rests on one independent source
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Open questions
How should readers weigh PSI against raw PM2.5 readings when the two can diverge, given that the Singapore study used PSI while the regional studies used PM2.5?
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How much of the child respiratory evidence from Indonesia and the asthma evidence from Thailand transfers to Singapore's population and pollution mix?
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What are the long-term health effects of repeated seasonal haze exposure, beyond the short-term associations documented so far?
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Why do regional mortality estimates for smoke haze vary so widely (roughly 1,064 to 260,000), and which figures are most reliable?
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