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Is road traffic noise linked to heart disease?

Long-term traffic noise is linked to a modestly higher risk of heart disease, and the public health impact comes from how many people are exposed.

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Covers: This page reviews evidence from epidemiological studies on the association between road traffic noise exposure and cardiovascular outcomes such as hypertension, ischaemic heart disease, and stroke. It does not cover occupational noise, aircraft noise, or clinical management of heart disease.

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The short answer

Evidence-backed AI-prepared starting map

Long-term exposure to road traffic noise is consistently associated with a modestly increased risk of cardiovascular disease in epidemiological studies. A meta-analysis of 11 prospective cohorts (1,353,481 participants) found higher traffic noise exposure associated with increased hypertension risk, both when noise was treated as a binary exposure (HR 1.07, 95% CI 1.01-1.13) and as a continuous exposure (HR 1.03, 95% CI 1.02-1.04). A systematic review and meta-regression of transportation noise reported an increase in myocardial infarction outcomes (RR 1.01, 95% CI 1.01-1.01). A narrative review reports associations between long-term traffic noise and arterial hypertension, ischaemic heart disease, myocardial infarction, heart failure and arrhythmias, often dose-dependent and remaining after adjustment for air pollution. In Ireland, environmental noise was estimated to account for 256 attributable cases of ischaemic heart disease and 3,445 IHD DALYs in 2017, with total noise-attributable burden of 14,091 DALYs exceeding that estimated for ambient air pollution.1234

What this rests on5 independent sources
  • Evidence 12
  • Interpretation 5

In brief

  1. Pooled cohort evidence links long-term traffic noise to higher hypertension risk, with hazard ratios of 1.07 (binary exposure) and 1.03 (continuous exposure) across 11 studies and 1,353,481 participants.1

    Evidence-backed
  2. A meta-regression of transportation noise found a small but statistically significant increase in myocardial infarction outcomes (RR 1.01, 95% CI 1.01-1.01).2

    Evidence-backed
  3. Associations are reported as dose-dependent and remain after adjustment for air pollution, and are supported by mechanistic evidence on endothelial function, stress responses and sleep disruption.3

    Evidence-backed
  4. At population level, environmental noise was estimated to account for 256 ischaemic heart disease cases and 3,445 IHD DALYs in Ireland in 2017, with total noise burden exceeding that of ambient air pollution.4

    Evidence-backed
  5. Individual-level risk increases are modest; the public health weight of the finding comes from widespread exposure rather than large per-person effects.14

    Interpretation

At a glance

The picture in numbers

Live · updated just now

Irish burden-of-disease assessment

256 cases

256 cases: ischaemic heart disease cases attributed to environmental noise in Ireland in 20174
Irish burden-of-disease assessment

3,445 DALYs

3,445 DALYs: ischaemic heart disease DALYs attributed to environmental noise in Ireland in 20174
Exceeded the burden estimated for ambient air pollution

14,091 DALYs

14,091 DALYs: total DALYs attributed to environmental noise in Ireland in 20174

The evidence behind it

5 sources
  • Reviews of many studies3
  • Other studies and data2

Published in 2025 and 2026

Sources on this page by kind and year
SourceKindYear
Exposure-response relationship between transportation noise and cardiovascular disease outcomes: a systematic review and meta-regression analysis.Reviews of many studies2026
Long-term exposure to traffic noise and the incidence of hypertension: a systematic review and meta-analysis of prospective cohort studies.Reviews of many studies2026
[Noise and the heart].Other studies and data2026
Association between long-term aircraft noise exposure and cardiovascular disease: a meta-analysis with focused subgroup analyses.Reviews of many studies2025
Estimated health burden of environmental noise exceeds that of ambient air pollution in Ireland.Other studies and data2026

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What it means for you

Which fits you?

Pick the situation closest to yours. Each answer says what it rests on.

If you live or sleep close to a busy road and are concerned about blood pressure

the pooled cohort evidence associates higher long-term traffic noise exposure with increased hypertension risk (HR 1.07 binary, 1.03 continuous), so it is reasonable to discuss blood pressure monitoring with a clinician and to consider practical exposure reduction such as quieter sleeping arrangements.1

Evidence-backed

If you already have or are at risk of ischaemic heart disease

the evidence links traffic noise to ischaemic heart disease and myocardial infarction, with associations reported as dose-dependent and independent of air pollution, so noise exposure is worth mentioning alongside other risk factors in clinical conversations.32

Evidence-backed

If you are making local or national environmental health policy

the Irish burden assessment estimated 256 ischaemic heart disease cases and 3,445 IHD DALYs attributable to environmental noise in 2017, with total noise burden exceeding that of ambient air pollution, supporting integration of noise into routine health risk assessment and noise reduction strategies.4

Evidence-backed

If you want to judge how much a single study should change your view

note that the pooled effect sizes are small (HR 1.03-1.07 for hypertension, RR 1.01 for myocardial infarction) and come from observational cohorts, so consistency across reviews and mechanistic plausibility matter more than any one estimate.123

Interpretation

If you are looking for evidence on aircraft or occupational noise

this page's scope is road traffic noise; aircraft noise findings are reported separately in the literature and should not be assumed to transfer directly to road traffic exposure.5

Interpretation

The full story · 2 chapters

01

What the evidence shows for road traffic noise and heart disease

AI summary:Pooled cohort and review evidence links long-term traffic noise to hypertension, heart attacks and other heart conditions, with population-level burden estimates.

Evidence-backed

Evidence-backed: The strongest pooled evidence concerns hypertension. A systematic review and meta-analysis of 11 prospective cohort studies with 1,353,481 participants found that higher traffic noise exposure was significantly associated with increased hypertension risk, with a hazard ratio of 1.07 (95% CI 1.01-1.13, P = 0.03) when noise was analysed as a binary variable, and 1.03 (95% CI 1.02-1.04) when treated as a continuous variable. The authors concluded that long-term traffic noise exposure is significantly associated with increased hypertension risk in adults and recommended incorporating noise assessment and mitigation into public health strategies.1

Evidence-backed

Evidence-backed: For cardiac events, a systematic review and meta-regression of transportation noise reported an increase in myocardial infarction outcomes with a relative risk of 1.01 (95% CI 1.01-1.01). The authors describe their work as the first to apply both conventional meta-regression and Burden of Proof methodologies to quantify associations between long-term transportation noise (road traffic, railway and aircraft) and combined fatal and non-fatal cardiovascular outcomes including myocardial infarction, ischaemic heart disease, stroke and ischaemic stroke, and propose new risk-outcome pairs for potential inclusion in Global Burden of Disease work.2

Evidence-backed

Evidence-backed: A narrative review reports that epidemiological studies consistently demonstrate associations between long-term traffic noise exposure and increased risk of arterial hypertension, ischaemic heart disease, myocardial infarction, heart failure and arrhythmias. It notes these associations are often dose-dependent and persist after adjustment for air pollution. Complementary experimental human studies show that even short-term noise exposure can impair endothelial function, activate autonomic stress responses and disrupt sleep. Proposed mechanisms include sympathetic nervous system activation, dysregulation of the hypothalamic-pituitary-adrenal axis, oxidative stress and inflammatory processes. The review concludes that traffic noise should be considered a modifiable cardiovascular risk factor, requiring both individual exposure reduction and population-level noise reduction strategies.3

Evidence-backed

Evidence-backed: At population level, an Irish burden-of-disease assessment estimated that in 2017 environmental noise exposure was associated with 256 attributable cases of ischaemic heart disease and 3,445 IHD DALYs, alongside 296,471 cases of high annoyance and 109,951 cases of high sleep disturbance. Total noise-attributable burden was 14,091 DALYs, exceeding the burden estimated for ambient air pollution and some dietary risks in comparable assessments. The authors describe environmental noise as a substantial but under-recognised public health risk and argue for stronger integration of noise into national environmental health governance and routine health risk assessment.4

Participant opinion · poll

How much road traffic noise do you experience where you live?

How much road traffic noise do you experience where you live?A lot — it regularly disturbs my sleep or daily activitiesSome — noticeable but manageableA little — occasional, rarely bothers meNone — my home is quiet
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02

How to read these numbers

AI summary:Individual risk increases are small, but widespread exposure makes the population impact large, and consistency plus mechanisms support the link.

Interpretation

Interpretation: The effect sizes here are small at the individual level. A hazard ratio of 1.03-1.07 for hypertension and a relative risk of 1.01 for myocardial infarction mean that any single person's extra risk from traffic noise is modest; the public health significance comes from the large number of people exposed, which is why the Irish assessment can attribute hundreds of ischaemic heart disease cases and thousands of DALYs to noise. Readers weighing personal risk should keep this distinction between individual and population risk in mind.124

Interpretation

Interpretation: The consistency across independent reviews, the dose-dependent pattern reported in the narrative review, and the persistence of associations after adjustment for air pollution all strengthen the case that the relationship is not simply an artefact of co-exposure to traffic-related air pollution. The mechanistic evidence on endothelial function, autonomic stress responses and sleep disruption offers a plausible biological pathway. None of this, however, converts observational associations into proof of causation.32

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  1. 1
    Long-term exposure to traffic noise and the incidence of hypertension: a systematic review and meta-analysis of prospective cohort studies.
    Frontiers in cardiovascular medicine (Xia et al.)Published May 28, 2026Checked Oct 4, 2026
    “The quality of the research studies was evaluated based on the Newcastle-Ottawa Scale methodologically. The association was estimated by data pooling with fixed-or random-effect models and estimated by calculating the HR and its 95%CI.Results11 cohort studies with 1,353,481 participants were included into the present research. The results indicated that in cohorts analyzing traffic noise as a binary variable, higher exposure was significantly associated with an increased risk of hypertension (HR = 1.07, 95% CI: 1.01-1.13, P = 0.03). This finding is highly consistent with the meta-analysis results where traffic noise was treated as a continuous variable (HR = 1.03, 95% CI: 1.02-1.04, P ConclusionLong-term exposure to traffic noise is significantly associated with an increased risk of hypertension in the adult population. Incorporating environmental noise assessment and mitigation measures into public health strategies can contribute to the comprehensive management of hypertension in the population.Systematic review registrationhttps://www.crd.york.ac.uk/PROSPERO/view/CRD420251270952.”
  2. 2
    Exposure-response relationship between transportation noise and cardiovascular disease outcomes: a systematic review and meta-regression analysis.
    Journal of exposure science & environmental epidemiology (Gonzato et al.)Published Apr 6, 2026Checked Oct 4, 2026
    “increase in myocardial infarction outcomes (RR = 1.01, 95%CI: 1.01-1.01, p-value SignificanceThis study adds to the existing literature by providing a comprehensive comparison of the association between long-term exposure to transportation noise sources and CVD outcomes using both conventional and BoP methodologies.Impact statementThis is the first study to apply the conventional meta-regression and Burden of Proof methodologies to systematically quantify and evaluate associations between long-term exposure to transportation noise sources (i.e., road traffic, railway, and aircraft) and combined risk of fatal and non-fatal cardiovascular disease outcomes, including myocardial infarction, ischemic heart disease, stroke, and ischemic stroke. The application of these two approaches to deriving exposure-response functions provides additional insights into the quantification of the burden of disease attributable to transportation noise. Our findings using the Burden of Proof framework on transportation noise and CVD outcomes advance the integration of an additional environmental risk factor and propose new risk-outcome pairs for potential inclusion in the Global Burden of Disease study.”
  3. 3
    [Noise and the heart].
    Herz (Hahad et al.)Published Jun 24, 2026Checked Oct 4, 2026
    “Cardiovascular diseases are among the most extensively studied outcomes in this context. Epidemiological studies consistently demonstrate associations between long-term exposure to traffic noise and an increased risk of arterial hypertension, ischemic heart disease, myocardial infarction, heart failure and arrhythmias. These associations are often dose-dependent and also remain even after adjustment for air pollution. Experimental human studies provide complementary mechanistic evidence, showing that even short-term noise exposure can impair endothelial function, activate autonomic stress responses and disrupt sleep. Underlying mechanisms include activation of the sympathetic nervous system, dysregulation of the hypothalamic-pituitary-adrenal axis, oxidative stress and inflammatory processes. Overall, the available evidence supports a stronger consideration of traffic noise as a modifiable risk factor for cardiovascular disease. In addition to individual-level measures to reduce exposure, population-based strategies for noise reduction are essential.”
  4. 4
    Estimated health burden of environmental noise exceeds that of ambient air pollution in Ireland.
    Discover public health (Faulkner & Murphy)Published Sep 2, 2026Checked Oct 4, 2026
    “Uncertainty and sensitivity were assessed by propagating parameter uncertainty in exposure-response functions and by testing the influence of key exposure-model assumptions on attributable outcomes.ResultsIn 2017, environmental noise exposure was associated with 256 attributable cases of ischaemic heart disease (IHD) and 3,445 IHD DALYs; 296,471 cases of high annoyance (HA) and 5,866 HA YLDs; and 109,951 cases of high sleep disturbance (HSD) and 4,780 HSD YLDs. In total, 14,091 DALYs were attributable to noise exposure, exceeding the burden estimated for ambient air pollution and some dietary risks in comparable population-health assessments.ConclusionEnvironmental noise represents a substantial yet under-recognised public health risk in Ireland. Harmful-effects estimation provides efficient, policy-ready outputs for END reporting and action planning, while BoD estimation supports cross-risk prioritisation in public health. The findings support stronger integration of noise within national environmental health governance and routine health risk assessment.”
  5. 5
    Association between long-term aircraft noise exposure and cardiovascular disease: a meta-analysis with focused subgroup analyses.
    European journal of preventive cardiology (Sebastian et al.)Published Dec 1, 2025Checked Oct 4, 2026
    “Statistical analysis was performed using RevMan 5.4 with a random-effects model, pooling outcomes as odds ratios (ORs) and mean differences (MDs) with 95% confidence intervals (CIs). The study protocol was pre-registered in PROSPERO (CRD42024598651). Our meta-analysis included 34 studies with a combined sample of ∼35.5 million adults aged 18 years and older, with follow-up durations ranging from 1 to 20 years. Pooled analysis revealed a statistically significant association between aircraft noise exposure and the risk of cardiovascular disease (CVD), with an OR of 1.1 (95% CI: 1.07-1.14, P ConclusionOur study demonstrates a significant association between aircraft noise exposure and increased risk of cardiovascular events and mortality, with stronger association during night-time exposure. The observed increase in systolic blood pressure and higher hypertension risk in males suggest early and sex-specific cardiovascular effects. These findings highlight the importance of mitigating chronic aircraft noise exposure to reduce its long-term cardiovascular burden.”

How it changed

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  1. Version 2Oct 4, 2026Live now

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Open questions

  • Which exposure metric best captures long-term road traffic noise risk - day-evening-night average levels, night-time levels, or another indicator - and how much does the choice change the estimated effect on heart disease?

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  • To what extent do the observed associations reflect a causal effect of noise rather than residual confounding by air pollution, socioeconomic position, or other correlated exposures?

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  • Is there a threshold below which road traffic noise carries no measurable cardiovascular risk, or does risk rise continuously with exposure?

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  • Do interventions that reduce road traffic noise exposure - such as barriers, insulation or traffic management - measurably lower cardiovascular risk, and over what time frame?

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  • Which groups are most susceptible to noise-related cardiovascular effects, and how do effects differ by sex, age, or pre-existing conditions?

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